Michael Goldstein Csaba Tóth, has Paleomedicina done any research on salt intake? After personal experimentation, observation of the experiences of some long term ZCers, and some basic reading of anthropology, I'm not convinced that salt is necessary on a carnivorous diet. I share this hypothesis as shared by L Amber O'Hearn: https://twitter.com/KetoCarnivore/status/911355933443342336
But I'd be curious to know what conclusions, if any, you have come to after so many paleo keto clinical trials.
RemoveBut I'd be curious to know what conclusions, if any, you have come to after so many paleo keto clinical trials.
Csaba Tóth For the active transport of glucose in the enterocytes (Sodium-dependent glucose cotransporters) sodium is needed. It is impossible to obtain it through the basal surface of the enterocytes. It's only possible from the lumen. Therefore, the amount of sodium=salt consumption is directly proportional to the amount of carbohydrate (glucose) consumed. If you eat much carbohydrate you also need high amount of sodium=salt. It's just that simple.There are no risks associated with salt consumption. But carbohydrates make us eat more salt.
Salt has an inportant role in physiological processes. Our clinical experience with patients on the paleolithic ketogenic diet show that:
1. The need for salt is associated with the amount of carbohydrate consumed (more losely with fructose and glucose)
2. Patients on the paleolithic ketogenic diet, who eat only small amounts of carbohydrate, do not wish for salt. They start to feel "normal" foods to salty and automatically decrease salt in their diet.
3. Ion levels in our patients (Na, K, Ca, Mg) are physiological.
4. Level of glucose and magnesium are inversely related. We are just about to publsh a study on this issue.
We do not provide a specific recommendation on salt given that it normally adjusts to physiological needs. Also, an elevated intake of salt does not have a negative health effect. Although it may elevate the volume of intravascular volumen and cardiac output but this does not result in a significant elevation of blood pressure. Instead, hypertension is caused by an increase in fructose intake. Fructose, besides causing an elevation in uric acid, results in reduced NO release, which normally results in vasoconstriction. As a consequence blood pressure increases.
Salt has an inportant role in physiological processes. Our clinical experience with patients on the paleolithic ketogenic diet show that:
1. The need for salt is associated with the amount of carbohydrate consumed (more losely with fructose and glucose)
2. Patients on the paleolithic ketogenic diet, who eat only small amounts of carbohydrate, do not wish for salt. They start to feel "normal" foods to salty and automatically decrease salt in their diet.
3. Ion levels in our patients (Na, K, Ca, Mg) are physiological.
4. Level of glucose and magnesium are inversely related. We are just about to publsh a study on this issue.
We do not provide a specific recommendation on salt given that it normally adjusts to physiological needs. Also, an elevated intake of salt does not have a negative health effect. Although it may elevate the volume of intravascular volumen and cardiac output but this does not result in a significant elevation of blood pressure. Instead, hypertension is caused by an increase in fructose intake. Fructose, besides causing an elevation in uric acid, results in reduced NO release, which normally results in vasoconstriction. As a consequence blood pressure increases.
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